Human N-acetyl-inositol-non-hydrolyzable carnosine is effective in cataract treatment because of its antioxidant effect. Oxidation is due to excessive reactive oxygen species, cells are damaged [1] phenomenon.
But the human body has an antioxidant activity that inhibits reactive oxygen species. Although it will play a full antioxidant effect when young, but with the increase of age this function will gradually decline. With the inevitable phenomenon of aging. This function once the recession can not be restored to its original state.
In fact, cataract is also due to the oxidation of the lens was damaged and the phenomenon of white turbidity. So that 90% of cataract is caused by aging.
Inhibit the oxidation of this oxidation function. It is called the natural amino acid of carnosine.
When the body produces this carnosine cataract can be suppressed. This diminishing function is the direct cause of cataracts. However, human use of N-acetyl-inosine · non-water-decomposing carnosine can supplement this carnosine. This is the antioxidant effect of human N-acetyl-inosine and non-water-decomposing carnosine.

Function:
1. protects epithelial cell membranes in the stomach and restores them to their normal metabolism;
2. acts as an antioxidant and protects the stomach from alcohol and smoking-induced damage;
3. anti-inflammatory properties and moderates production of interleukin-8;
4. adheres to ulcerations, acts as a barrier between them and stomach acids and helps to heal them;
5. inhibits activity of Helicobacter pylori bacteria;
6. does not interfere with the production of prostaglandins essential to the mucosal lining of the stomach;
7. strengthens these mucosal linings and stimulates production of protective mucous.